Haass lab
@Haass_Lab
The Haass lab Twitter account | 🇩🇪 @DZNE_de @SyNergy_Cluster | 🔬AD, FTLD & microglia research | 🕵️ PI Christian Haass (signed -CH) | 💬 Tweets by @LisdeW_
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https://www.biochemie.abi.med.uni-muenchen.de/index.html 04-03-2021 12:02:28
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What Alzheimer noted as fat saccules in glia may have been lipid droplets. They can be induced by Abeta in microglia leading them to release factors that damage neurons. ApoE4 makes things worse rdcu.be/dA6Kh. Congrats Michael Haney Robert Palovics and fantastic team
My lab recently moved to Universität München joining the excellent environment Biomedical Center, LMU Munich and Munich Cluster for Systems Neurology (SyNergy). We are now looking for a #DataScientist with an interest in neuroepigenomics to support our team. The position is a tenure track professorship: tinyurl.com/NeuroepiProf Pls RT!
We are in #Munich today shooting the video portrait of Hector Fellow Christian Haass, Prof. for Metabolic #Biochemistry Universität München and speaker of Forschungszentrum DZNE | @[email protected] & Munich Cluster for Systems Neurology (SyNergy)! Young Researcher Matteo Rovere will also be part of it. We will let you know when it's out! Haass lab
Commentary on study in Nature Medicine on potential transmissibility of #Alzheimer 's⏩dzne.de/en/im-fokus/me…⏩No risk for infection in everyday contact with patients 🧠 Haass lab Mathias Jucker /mn
🎙Via ZEIT ONLINE WISSEN 🧠 Christian Haass Haass lab und Wolfgang Hoffmann über das #Alzheimer -Medikament #Lecanemab und die Versorgung von Menschen mit #Demenz ⏭️ zeit.de/gesundheit/202… /mn
Excited to share our new paper, Trem2 Promotes Foamy Macrophage Lipid Uptake and Survival in Atherosclerosis. Out today in Nature Cardiovascular Research! Great collaboration with Konstantin Zaitsev CFI-UMN UMN Integrative Biology & Physiology Ty Patterson
nature.com/articles/s4416…
Loss of TREM2 exacerbates parenchymal amyloid pathology but diminishes CAA in Tg-SwDI mice biorxiv.org/cgi/content/sh… #biorxiv_neursci
Very happy to share our new work in JAMA Neurology, showing that ApoE4 triggers earlier and faster amyloid-related tau spreading. Thus, Anti-amyloid therapies may need to come earlier in E4 carriers to prevent tau. Huge thanks to Anna Steward and my team!
doi.org/10.1001/jamane…