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📢 NEW PREPRINT | Discover how alternative splicing at the OAS2 gene diversifies antiviral profiles The shorter p69 isoform targets HCoV-OC43, while the longer p71 isoform restricts EMCV. Mechanistic plasticity reveals novel antiviral mechanisms. biorxiv.org/content/10.110…

Why can't these viruses be friends? In a new study from her PhD MRC-Uni of Glasgow Centre for Virus Research , Anna Sims shows that #SARSCoV2 infections segregate into distinct microdomains as they spread. But why? (1/N) journals.asm.org/doi/10.1128/jv…

I can't believe my second first-author paper has been published today, on my actual birthday! Double celebrations tonight 🥳🥳 Available to read here: journals.asm.org/doi/10.1128/jv…

Congrats Anna Sims on your second 1st author paper! Working alongside you was a highlight of my time in Glasgow and it's so special to be one of your coauthors!

Sooo happy to finally have this one published!! (3 years in the making… 👀) A comprehensive investigation into the recency and geographical origins of both SARS-CoVs that spilled to humans in the past few decades, now in Cell cell.com/cell/fulltext/…

Can flu spread to another cell without ever exiting the first one? Yes! Through little tunnels (TNTs) that form between cells! Check out Daniel's exciting paper and beautifully animated thread below to find out more!!! 🔬🔬🧫🧫

🦠 Influenza's Great Escape New research from Daniel Weir & colleagues reveals that flu viruses use secret tunnelling nanotube-like structures to escape your immune system. Full video: youtu.be/Opfpwy9yUew Read: gla.ac.uk/research/az/cv… Hutchinson Lab UofG Sii UofG MVLS